Erythrocytes

The complete aerobic oxidation of glucose to $CO_2$ and water takes place through the Krebs cycle and the electron transport system, which are located in the mitochondria.
Mature mammalian red blood cells lack a nucleus, mitochondria and other cell organelles; this leaves more room for haemoglobin.
Since they do not have mitochondria, the red blood cells cannot carry out the Krebs cycle.
They obtain their energy only by anaerobic glycolysis, in which glucose is converted to lactic acid, so they respire only anaerobically.
This also ensures that they do not use up the oxygen they carry.
Unstriated muscle cells, liver cells and white blood cells all have mitochondria and can oxidise glucose aerobically.
So the cells that cannot metabolise glucose to carbon dioxide aerobically are the red blood cells.

Glucose-6-phosphate dehydrogenase (G6PD) is the first enzyme of the pentose phosphate pathway, which produces NADPH.
In red blood cells this pathway is the only source of NADPH, which keeps glutathione in its reduced form; reduced glutathione protects the cells against damage by oxidising agents.
In G6PD deficiency (an X-linked recessive disorder) the RBCs cannot protect themselves from oxidative stress.
On exposure to certain drugs (such as primaquine and sulphonamides), infections or fava beans, the haemoglobin and membranes of the RBCs are damaged and the cells burst.
This haemolysis of red blood cells causes haemolytic anaemia.
So G6PD deficiency is associated with the haemolysis of RBCs.